However, in the context of IPF, it has been confirmed that there are some dysplastic P63+ progenitor cells distributed near the fibrotic foci, which might be pro-fibrotic with potential to exacerbate the IPF disease (Habermann et al., 2020; Jaeger et al., 2022; Wang et al., 2023). This evidence concerns the gene TP63 and idiopathic pulmonary fibrosis.