Relatedly, while deploying HS or heparin as decoy viral receptors has been proposed as a way to inhibit viral spreading in patients47,48, their delivery could also have the effect of binding up reparative HSBPs such as Areg during tissue recovery; thus, further work is needed to investigate the ideal temporal deployment of these molecules to curtail viral infection while ensuring proper tissue repair. This evidence concerns the gene AREG and viral infectious disease.