At 3d and 7d post-MI/R, FAP+ myofibroblasts and CD68+ macrophages were located in proximity within infarcted myocardium, but remained separate cell populations (Fig. 1C, D), underscoring that myofibroblast activation and monocyte recruitment are distinct mechanisms during the wound healing phase post-MI/R. Here, CD68 is linked to myocardial infarction.