In particular, mice treated with the bacteria from the e2 donor, regardless of the transplantation protocol, showed increased levels of molecules released in response to infections and tissue injuries (IL6, TNFα, IL1ß, IL12p70) as well as involved in the regulation of migration and infiltration of neutrophils (MIP1α, GROα), eosinophils (eotaxin), T-cells (IP10) and monocytes/macrophages (MCP1, GMCSF, MCP3) (Figures 4A–E, respectively). The gene discussed is CXCL10; the disease is infection.