The rapid increase in serum sodium concentration can be held accountable for osmotic demyelination which led to the neurologic symptoms [27] and the thrombosis of the femoral artery can be attributed to a hypernatremia–hyperosmolality-induced hypercoagulability due to an increase in endothelial production and secretion of von Willebrand factor [28]. Here, VWF is linked to Hypernatremia.