AMH inhibits FSH-stimulated granulosa cell aromatase activity (Grossman et al. 2008), and the resulting hyperandrogenemia increases expression of FSH receptors, leading to follicle recruitment and pre-antral follicle growth (Luo & Wiltbank 2006, Catteau-Jonard et al. 2008). This evidence concerns the gene BRD2 and polycystic ovary syndrome.