AKT1 and neoplasm: Additionally, the presence of m5C‐modified mRNAs in tumour‐related pathways, including the Phosphoinositide 3‐Kinase‐Protein Kinase B Signaling Pathway (PI3K‐AKT) and Extracellular Signal‐Regulated Kinase‐Mitogen‐Activated Protein Kinase (ERK‐MAPK) signalling pathways, suggests that m5C hypermethylation may instigate tumourigenesis by activating oncogenic signalling pathways.129