Wan et al. (2019) demonstrated that high‐concentration Chang Wei Qing remarkably decreased tumor size and quantity, and inhibited NF‐κB and STAT3 pathways to retard colitis‐related tumor progression, which mainly contributed to the potential benefits of Chang Wei Qing in drug‐caused colitis‐related cancer auxiliary regulation. This evidence concerns the gene NFKB1 and neoplasm.