The same authors later demonstrated that in other glioma cell lines (U118MG and T98G), treatment with CBD (10 μM), isolated or combined with irradiation (10 Gγ), and the ATM kinase inhibitor KU60019 (1–2 μM) induced apoptosis through the upregulation of TRAIL/TRAIL-R2 and promoted DR5 activation [36]. The gene discussed is TNFRSF10B; the disease is central nervous system cancer.