Supporting these findings, other authors showed increased densities of CD57-enriched NK cells within pulmonary tissue and lymphoid follicles in lungs of severe COPD patients, suggesting that, following inflammatory stimulation as seen in COPD, NK cells migrate from the bloodstream to the lungs, where they may contribute to COPD-related pathologies [27,28]. This evidence concerns the gene B3GAT1 and chronic obstructive pulmonary disease.