Previously, it has been shown that IL-17A is a pro-inflammatory cytokine [49], stimulating the recruitment of other immune cells into insulitis and the progression of T1D [50], which might be the background for our findings and a potential mechanistic link, implicating the role of IL-17 in starting and promoting inflammatory attack in subjects at higher risk of T1D development. The gene discussed is IL17A; the disease is type 1 diabetes mellitus.