Atopic asthma is characterized by a Th2-skewed immune response, marked by the activation of Th2 cells that release cytokines such as Interleukin 4 (IL-4), IL-5, and IL-13, which can lead to eosinophilia, mucous production, respiratory inflammation, and airway remodeling [14]. This evidence concerns the gene IL4 and atopic asthma.