In summary, the protein targets identified in Method 2 offer valuable insights into the molecular mechanisms involved in AD + P. By focusing on key proteins, such as integrins, tyrosine hydroxylase, calcium channels, 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase, and gamma-aminobutyric acid (GABA) receptors, we can begin to uncover the intricate pathways that may contribute to both the onset and progression of psychosis in Alzheimer’s disease. This evidence concerns the gene TH and psychotic disorder.