To ascertain whether, as with other P1B-type ATPases, CtpA may be involved in the intracellular survival of Mtb [20,28], the bacterial load (CFU/mL) was quantified at different post-infection times in mouse alveolar macrophages (MH-S cell line) infected with the wild-type MtbH37Rv or mutant MtbΔctpA strain at MOI 1:3 (Figure 3). Here, EPHA2 is linked to infection.