While these results support the hypothesis that uPTM-FetA excretion is partly related to obesity and metabolic syndrome variables [15], a lack of association with eGDR, adiponectin, and leptin, and the weak effect size of the linear regression model indicate that, in this cross-sectional study, other unidentified factors determine uPTM3-FetA excretion in patients with T1D, normoalbuminuria, and preserved kidney function. This evidence concerns the gene ADIPOQ and obesity disorder.