KIF18B and neoplasm: In the same study, gene set enrichment analyses of TCGA LUAD tumours with high versus low KIF18B mRNA expression suggested KIF18B-high tumours were enriched for gene expression signatures indicative of mTORC1 signalling, G2/M checkpoint, MYC, E2F, and mitotic spindle regulation pathways, consistent with KIF18B’s roles in mitosis and its correlation with a proliferative capacity [44].