In this study, the task was to develop a comprehensive mathematical model that accounts for various post-myocardial infarction (MI) events and factors, including the inflammatory dynamics of γδT cells, IL–17 elements, neutrophils, and ROS-mediated ischemia-reperfusion (IR) injury, cardiac remodeling driven by necrosis and fibrosis, and stem cell therapy. The gene discussed is IL17A; the disease is myocardial infarction.