In addition to increased non‐ischemic hydrogen peroxide levels in response to cytochrome c (9.88 ± 4.59 vs. 6.52 ± 2.61 pmol·s−1·mg−1; p = 0.005), male CRS rats produced elevated levels through β‐oxidation (11.23 ± 8.68 pmol·s−1·mg−1 vs. 6.71 ± 2.84 pmol·s−1·mg−1; p = 0.020) in non‐ischemic tissue relative to controls (Figure 2). This evidence concerns the gene CYCS and congenital rubella syndrome.