Finally, a similar study identified miRNAs linked to mucin-type O-glycan biosynthesis, such as miR-210-5p, introducing a less-reviewed pathway associated with excessive mucus production in CRSwNP, while downregulated miRNAs, including miR-32-3p, targeting TGF-β and MAPK signaling were reportedly linked in tissue remodeling and inflammation [49]. The gene discussed is TGFB1; the disease is chronic rhinosinusitis with nasal polyps.