Additionally, we assayed peptides typically phosphorylated by the AXL (Tyro3‐Axl‐Mer (TAM) receptor) tyrosine kinase, the insulin (InSR) and insulin‐like growth factor 1 receptor (IGF1R), the ALK, neurotrophic TK, the kit proto‐oncogene and the fibroblast growth factor, and we obtained clear evidence for their increased activity in tumours of LC patients (range in AD 2–4‐fold, in SQ 2–24‐fold). The gene discussed is ALK; the disease is laryngotracheoesophageal cleft.