reported that METTL14 inhibited aerobic glycolysis in p53 wild‐type CRC by downregulating GLUT3 and PGAM1 expression through the selective enhancement of the m6A‐YTHDF2–dependent processing of pri‐miR‐6769b and pri‐miR‐499a [46]. Here, PGAM1 is linked to colorectal carcinoma.