A recent study reported ﻿that ﻿the transcript levels of HERV‐K(HML‐2) gag, pol, and env genes were significantly upregulated in CRC [43], but further comparison with the present study is prevented by the fact that authors used probes and primers with unknown range of specificity among the around 90 HML2 elements present in the human genome [43]. The gene discussed is ERVW-1; the disease is colorectal carcinoma.