PSRC1 and atherosclerosis: These HDL measurements all showed strong colocalization with CAD and IS, the two major ASCVD subtypes, but at totally divergent genetic loci, and subsequent integration with multi‐tissue eQTL data revealed causal association as well as colocalization with CAD only of PSRC1 expressed in whole blood at the shared causal variant rs7528419, indicating therapeutic potential for CAD.