Moreover, the presence of higher NK cell abundance and dendritic cell activity in low‐risk patients suggests an active engagement of the innate immune system, which could be leveraged for therapeutic interventions.[30, 31, 32] The identification of NOX1 as a differentially expressed gene further highlights its role in gastric cancer biology, with functional assays indicating that NOX1 promotes cancer cell proliferation, migration, and invasion, making it a potential target for future therapies.[33, 34]. This evidence concerns the gene NOX1 and cancer.