We first analyzed the molecular signatures of resected tissues from six CRC patients, in which the tumoroids from patient #3 exhibited high levels of E‐cadherin and phosphorylated β‐catenin, while patient #6 exhibited low levels of E‐cadherin and phosphorylated β‐catenin, but high levels of myosin activity, CD31, and CD61 (integrin β3), representative of CRCs with epithelial and hybrid EMT state, respectively (Figure 8E,F; Figure S15B, Supporting Information). This evidence concerns the gene MYH14 and colorectal carcinoma.