As downregulation of GSTA4 in VAT of mice was found to increase 4‐HNE‐mediated protein carbonylation, oxidative stress, and mitochondrial dysfunction to exaggerate obesity and insulin resistance,[19] the expression of GSTA4 and the abundance of 4‐HNE‐modified protein and carbonylated protein in eWAT were also determined. The gene discussed is GSTA4; the disease is Obesity.