GCGR and steatosis: In the non‐fasted state, liver TG concentrations were increased in female Gcgr−/− mice (p = 0.02), whereas in the fasted state liver TG concentrations were similar in Gcgr−/− and Gcgr+/+ mice (p = 0.7) but increased compared to the non‐fasted state (Figure 3e), suggesting that fasting induced‐steatosis masks the steatosis‐promoting effect of permanent genetic deletion of the glucagon receptor.