A previous study reported that injury and functional biomarkers such as tissue inhibitor of metalloproteinase 2 (TIMP-2), insulin-like growth factor-binding protein 7 (IGFBP7), neutrophil gelatinase-associated lipocalin (NGAL), and L-type fatty acid-binding protein (L-FABP) could be used in combination with clinical information to enhance the ability to diagnose AKI, identify different pathophysiological processes, differentiate the aetiology of AKI, and assess AKI severity [18]. Here, LCN2 is linked to acute kidney injury.