,13 To explore whether HDV infection may also favour the hepatic recruitment of antigen-specific and eventually also of non-antigen-specific immune cells expressing CXCR3, we comparatively analysed a cohort of HBV-mono-infected or HDV-super-infected mice that had been adoptively transferred with blood-derived human T cells expressing either an HBV-specific TCR or an infection-unrelated TCR.12 This evidence concerns the gene CXCR3 and infection.