A recent study showed that cholecystokinin-expressing (CCK) vestibular nucleus (VN) neurons elicit autonomic responses in MS after activation, suggesting that vestibular function training may alleviate subsequent motion sickness symptoms by stimulating vestibular organs to a certain extent, leading to changes in the central vestibular nuclei and CCK-expressing VN neurons, which in turn alleviates subsequent motion sickness symptoms (27). This evidence concerns the gene CCK and myeloid sarcoma.