2023; Matter et al. 2024). Building on the previous findings that QXJYG improves the prognosis in a myocardial infarction mouse model, our study further demonstrates that the suppression of cardiac inflammation may be a key mechanism underlying this protective effect (Chen et al. 2024). Through in vivo and in vitro experiments, we further elucidated that the beneficial effects of QXJYG on reducing post-myocardial infarction and hypoxia-induced inflammation may be mediated via the regulation of the MK2/TTP signaling pathway. The gene discussed is ZFP36; the disease is myocardial infarction.