Another study revealed that METTL1 increases serine/arginine‐rich splicing factor 9 (SRSF9) expression in a m7G‐dependent manner, facilitating alternative splicing and the stabilisation of nuclear factor of activated T cells, cytoplasmic, calcineurin‐dependent 4 (NFATc4) and thus promoting cardiac hypertrophy.62 Here, SRSF9 is linked to cardiac hypertrophy.