In contrast to what was found for ataxia telangiectasia, it is mostly accepted that low lamin B1 is a senescence biomarker, e.g., Dreesen et al. on how lamin B1 declines with senescence (Dreesen et al. 2013), and Freund et al. on how if irradiation decreases lamin B1, a senescent phenotype follows (Freund et al. 2012; Kristiani and Kim 2023). The gene discussed is LMNB1; the disease is ataxia telangiectasia.