This process is essential for the concentration and functional regulation of biological macromolecules, including proteins and RNA.[12, 13] LLPS has been widely observed to regulate critical cellular processes in cancer cells through the formation of biomolecular condensates, which fundamentally disrupt cellular homeostasis and promote tumorigenesis.[14, 15] For instance, DAZAP1 promotes COX16 expression via LLPS, thereby driving tumor growth and metastasis.[16] However, the precise connection between molecular condensates and cancer cell pathophysiology remains elusive. The gene discussed is COX16; the disease is cancer.