demonstrated that the LLPS of nuclear YBX1, enhanced by circASH2, accelerates the decay of TPM4 transcripts.[50] Additionally, MELTF‐AS1 can drive the phase separation of YBX1, thereby activating ANXA8 transcription and promoting tumorigenesis in non‐small cell lung cancer.[51] These studies suggest that the LLPS of YBX1 within the nucleus is essential for transcriptional regulation. Here, MELTF is linked to lung cancer.