The expression level of circSnx12 is upregulated in cisplatin-resistant ovarian cancer tissues compared to cisplatin-sensitive ones; circSnx12 knockdown increases the chemosensitivity of ovarian cancer to cisplatin both in vitro and in vivo, inhibits cell proliferation, stimulates apoptosis, decreases GSH, increases lipid peroxidation, and activates ferroptosis in ovarian cancer via the circSnx12/miR-194-5p/SLC7A11 (Qin et al., 2023[103]). Here, SLC7A11 is linked to ovarian carcinoma.