Given that the correlation between pathological β power and SNpc dopaminergic neuron count has been demonstrated primarily in PD toxin models, but not in human PD,3 and considering conflicting outcomes of striatal dopamine transporter imaging in relation to SN neuron numbers in PD patients,46,47 our study suggests a translationally relevant method for predicting SNpc neuron loss in PD by combining measurements of low β oscillations with assessments of striatal dopaminergic levels. This evidence concerns the gene SLC6A3 and Parkinson disease.