Almost all of them have well-known relationships with GBM pathogenesis, e.g., aryl hydrocarbon receptor (AHR) [55], forkhead box M1 (FOXM1) [56], hypoxia inducible factor 1 subunit alpha (HIF1A) [57], Jun proto-oncogene (JUN) [58], MYC proto-oncogene (MYC) [59], nuclear factor kappa B subunit 1 (NFKB1) [60], signal transducer and activator of transcription 1 and 3 (STAT1, STAT3) [61], [62], etc. The identification of TFs, well studied in GBM, confirms the reliability of the obtained results. This evidence concerns the gene STAT3 and glioblastoma.