Most of them have well-known relationships with GBM pathogenesis, e.g., hypoxia inducible factor 1 subunit alpha (HIF1A) [57], Jun proto-oncogene, AP-1 transcription factor subunit (JUN) [58], MYC proto-oncogene, bHLH transcription factor (MYC) [59], nuclear factor kappa B subunit 1 (NFKB1) [60], signal transducer, and activator of transcription 1 and 3 (STAT1, STAT3) [61], [62]. Here, NFKB1 is linked to glioblastoma.