Following myocardial infarction, Cardiac-Fibroblasts assume a critical role in myocardial scar repair.[56] Meanwhile cardiac fibroblasts are closely associated with ventricular remodeling and post-infarction heart failure.[57,58] Scholars have investigated signaling pathways linked to this theme using diverse omics methodologies, such as genome, transcriptome, and proteome, confirming the involvement of inflammatory cytokines TGF-β,[59,60] RAAS,[61] several miRNAs,[62,63] and other regulatory factors in cardiac fibroblast regulation. Here, TGFB1 is linked to myocardial infarction.