Nevertheless, previous study reported that MT1A tranduced into the mitochondria of MPP+-treated SH-SY5Y neuroblastoma cells was able to alleviate mitochondrial damage, as shown by restored ATP levels, mitochondrial NADH dehydrogenase activity and mitochondrial superoxide levels (Kang et al., 2018). This evidence concerns the gene ENSG00000251605 and neuroblastoma.