NAA15 and coronary artery disorder: Mechanistic studies usinghuman isogenic induced pluripotent stem cells (iPSCs) derived from CHD patients withNaa15 variants (Naa15+/− iPSC-derived cardiomyocytes) revealed alteredexpression levels in 562 out of 650 NatA-targeted proteins, suggesting a role ofNatA-mediated N-terminal acetylation in protein stability[228].