Lastly, to define whether inactivation of LATS1/2 and activity of YAP/TAZ/TEADs correlate with similar stromal phenotypes in human breast cancer, we used TIMER (Li et al, 2020), which is a computational platform that estimates stromal cell infiltration based on RNA-seq data from The Cancer Genome Atlas (TCGA). This evidence concerns the gene LATS1 and breast carcinoma.