The results showed that HA‐tagged full‐length PLK1 in PLK1 inhibitor BI2536‐treated PCa cells was still able to interact with SHCBP1, while that in PBD inhibitor poloxin‐treated cells were unable to interact with SHCBP1 (Figure 6F). The gene discussed is SHCBP1; the disease is posterior cortical atrophy.