In vitro, knocking down Kif3a increased epithelial apoptosis, boosted CCL17, IL‐5, and IL‐8 transcription, and raised COX‐2 protein levels and β‐catenin translocation; Conversely, Kif3a overexpression greatly alleviates asthma (Geng et al. 2018). The gene discussed is KIF3A; the disease is asthma.