In vitro, knocking down Kif3a increased epithelial apoptosis, boosted CCL17, IL‐5, and IL‐8 transcription, and raised COX‐2 protein levels and β‐catenin translocation; Conversely, Kif3a overexpression greatly alleviates asthma (Geng et al. 2018). This evidence concerns the gene CXCL8 and asthma.