(14) reported that the combination of HHT and BTZ influenced the proliferation of SKM-1 cells, a myelodysplastic syndrome (MDS) cell line, by interfering with the AKT and NF-κB signaling pathways, upregulating the expression of their downstream oncogene p53, and decreasing the expression of miR-3151. This evidence concerns the gene NFKB1 and myelodysplastic syndrome.