Through conducting Western blot, we found that the levels of β-catenin and p-AKT, as well as the downstream factors, including c-Myc and EpCAM, were decreased with the knockdown of SLC1A4 in Huh7 and HepG2 cells (Figure 2c), indicating the potential mechanism regulated by SLC1A4 in HCC. The gene discussed is SLC1A4; the disease is hepatocellular carcinoma.