The authors suggested iNOS “as a marker of deleterious inflammatory reactions”; and (vi) a recent report on a model of Alzheimer’s disease induced in rats by aluminium chloride caused the augmentation of cerebellar iNOS protein expression in Purkinje cells, which was inhibited by the flavonoid naringin [41]. The gene discussed is NOS2; the disease is early-onset autosomal dominant Alzheimer disease.