Interestingly, we found a significant increase in the expression of mitochondrial fusion proteins, including mitofusin 1 and 2 (MFN1/2) and optic atrophy (OPA), in OECM‐1 CSCs after 6 and 16 h hypoxia exposure compared with similarly treated OECM‐1 cells. This evidence concerns the gene MFN1 and Leber hereditary optic neuropathy.