IL‐1ꞵ, IL‐6, and TNF‐α have, in turn, exhibited potency in influencing cardiac electrical remodeling, as evidenced by prolonged action potential duration (APD) and QT intervals (markers of delayed repolarization) (Chowdhury et al. 2021; A. Corbin et al. 2014a, 2024b) and increased incidence of early after‐depolarizations (EADs) (Chowdhury et al. 2021), which are indicative of a reduced repolarization reserve and are potential causes of lethal VAs such as torsade de pointes (TdP) (Weiss et al. 2010). This evidence concerns the gene IL1B and torsades de pointes.