Further investigations found several factors, such as the epidermal growth factor receptor (EGFR), phosphoinositide 3-kinase (PI3Ks), p21-activated kinase-1 (PAK1), Niemann-Pick C1 (NPC1), and NPC2, as potential factors in ASFV entry, highlighting the complexity of ASFV infection mechanisms [172,173,174]. The gene discussed is EGFR; the disease is infection.