In this cohort, composed of 116 patients with chordoma NOS, 14 with conventional chordoma, and 3 with dedifferentiated chordoma, mutations in SWI/SNF complex genes (PBRM1, ARID1A, SETD2) were not significantly enriched or depleted across subtypes (p > 0.05). Here, PBRM1 is linked to chordoma.